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Mouse brain white matter. White matter injury in mouse brain yaning li a guanghui tang a yanqun liu b xiaosong he a jun huang a xiaojie lin a zhijun zhang a guo yuan yang a b yongting wanga key words. I optogenetic stimulation of the mouse acc in the range of 1 8 hz results in changes to white matter in the corpus callosum. The brain white matter contains millions of nerve fibers or axons that connect other parts of the brain and spinal cord and signal your nerves to talk to one another. Here applications of diffusion tensor imaging in detecting major and subtle white matter pathology in the mouse cns are reviewed followed by several technical details that may be helpful in designing studies that involve diffusion tensor imaging of rodent brain and spinal cord.
Ii this low frequency stimulation results in increased oligodendrocyte proliferation accompanied by a decrease in the g ratio within the corpus callosum underlying the acc. More damage from the white matter injury was observed in amd3100 treated mice than in the aav gfp group at 3 weeks of ischemia. Our results can be summarized as follows. This data set shows the projections and projection topographies from given anatomical structures.
C x c chemokine ligand 12 x ischemia x oligodendrocyte progenitor cell x remyelination x white matter abstract. Around 40 of the human brain consists of white matter which mainly contains myelinated axons that allow rapid transmission of action potentials to distant brain areas 1 2. Blocking cxcl12 cxcr4 signaling pathway with amd3100 abrogated the beneficial effect of cxcl12 gene therapy in protecting striatum white matter integrity at 5 weeks after ischemia and decreased the number of pdgfrα opcs in the svz and perifocal area in the 3 week ischemic brain. Diffusion tensor imaging has been increasingly used for studying white matter pathology in rodent models of neurological diseases.
Delineate gray matter and white matter structures in 3 d. Methods for data generation have.
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